Pulp tissue rich in vascular, neural and odontoblast cells; have the ability to conduct defensive reaction, namely the ability to hold the recovery in the event of inflammation. However, in case of chronic inflammation in the pulp tissue or an advanced process of pulp tissue inflammation will cause the death of the pulp / pulp necrosis. It is as a result of the failure of the pulp tissue in seeking recovery or cure. The more extensive damage to the inflamed pulp tissue weight of the rest of the healthy pulp tissue to maintain its vitality.
Pulp necrosis occurs basically started because of a bacterial infection in the pulp tissue. This can occur due to contact between the pulp tissue in the oral environment due to the formation of dentinal tubules and pulpal direct exposure, it facilitates the infection of bacteria into the pulp tissue that causes inflammation of the pulp tissue. If treatment is not done, then the inflammation of the pulp will get worse and may occur changes in blood circulation in the pulp, which in turn lead to pulp necrosis. Dentinal tubules can be formed as a result of the operative or restorative procedure or due to poor restorative material that is irritating. It could also be caused by fracture of the enamel, dentin fracture, the process of erosion, attrition and abrasion. Dentinal tubules of this bacterial infection can reach the pulp tissue and cause inflammation. While direct pulpal exposure can be caused by the trauma, operative procedure and the most common is due to the presence of caries. This resulted in the pulp tissue and bacteria infect the pulp tissue inflammation.
Pulp necrosis is caused by trauma to the teeth can lead to pulp necrosis in the immediate few weeks. Basically the same process that occurs in the blood circulation changes in the pulp, which in turn lead to pulp necrosis. Trauma to the teeth can cause obstruction of the main blood vessel in the musty and subsequently resulted in the dilation of capillaries in the pulp. Pulpal capillary dilatation was followed by capillary degeneration and edema pulp. Due to lack of collateral circulation in the pulp, it can occur in part or total myocardial ischemia in the pulp and causing pulpal response to low inflammation. This allows the bacteria to penetrate down to the small vessels of the virgin at the apex. All these processes can result in pulp necrosis.
