Pulp necrosis is the death of the pulp which is a continuation of the inflammatory process of the pulp acute / chronic or cessation of blood circulation caused by sudden trauma. Pulp necrosis may occur parsialis or totalis3.There are two types of pulp necrosis, namely:
A. type of coagulation
In this type there is a part of the dissolved, precipitated and transformed into a solid material.
2. type of liquefaction
In this type, proteolytic enzymes modify pulp tissue into a soft material or cair3.
At each process pulp death always produced the final form of H2S, ammonia, materials that are fat, indikan, protamain, water and CO2. Among them are also produced indole, skatol, putrescine and kadaverin, causing a stench in the death of the pulp. When the events of necrosis also enter saprophyte germs are anaerobic, the death of the pulp is called gangrene pulpa3.
Necrosis or death of the pulp has a variety of causes, usually due to inflammation of the state of irreversible pulpitis is no treatment or can occur suddenly due to traumatic injury that interferes with blood supply to the pulp. Although the remaining pulpal necrosis of melted or dikoagulasikan, pulp continue to experience death. Within a few hours of the inflamed pulp may degenerate into a condition nekrosis2. Other causes of nekrosi bacteria, trauma, irritation of silicate restorative materials, or acrylic. Pulp necrosis may also occur in the application of materials such as arsenic and paraformaldehid4 devitalisasi.
Pulp necrosis can occur rapidly (within weeks) or a few months to chronic. Attrition and caries conditions are not handled well can lead to pulp necrosis. Pulp necrosis occurs more often in conditions of chronic phase than the acute phase
